Page 120 - 2014_SpringSale

Page 120

The Willner Window Product Reference Catalog, Spring 2014

since 1911

• Willner Chemists •

the nutritional supplement professionals

Source Naturals

Progress in Alzheimer’s: Vitamin

E and Omega-3’s

Vitamin E preserves activities of

daily living

In this study across 14 Veterans Affairs

Medical Centers, 561 people with mild to

moderate Alzheimer’s disease took 2,000 IU

of vitamin E per day, 20 mg per day of the

memory-loss drug memantine; these two

together, or a placebo. After more than two

years of follow-up, compared to placebo,

doctors found vitamin E delayed the decline

in daily functioning activities by 19 percent,

or by more than six months.

Researchers also looked at changes in the

amount of caregiver time and found those

who took vitamin E alone had the smallest

increase in time required of caregivers.

This is the first clinical trial to show an

added benefit for vitamin E in mild to mod-

erate Alzheimer’s disease and, because vita-

min E is inexpensive and readily available,

should be part of treatment, doctors said.

Reference: Journal of the American Medical

Association; 2014, Vol. 311, No. 1, 1810379

Omega-3s slow decline

The brain requires omega-3s and other

essential fatty acids, and those with

Alzheimer’s disease have lower-than-normal

concentrations in the brain. In this study, 33

men and women with mild Alzheimer’s dis-

ease took 2,300 mg of high-DHA omega-3s

per day, or a placebo. After six months, while

the placebo group had not changed, those

taking omega-3s had higher levels of DHA

and EPA in the blood and in cerebrospinal

fluid.

The cerebrospinal fluid finding is important

in humans, doctors said, because before now

they had not known if omega-3 supplements

could cross the blood-brain barrier, a mem-

brane network that separates the brain from

the circulatory system and protects the cen-

tral nervous system from harmful chemicals

while allowing key nutrients to pass through

and nourish the brain.

Doctors also found that as the levels of

DHA increased, signs of Alzheimer’s disease

and inflammation decreased, suggesting

omega-3s may be helpful in preserving

healthy cognitive and physical function.

Reference: Journal of Internal Medicine;

January, 2014, Published Online

Nutrients help preserve brain

function and memory

Maintain Brain Health

What are telomeres?

Cells contain a string of DNA with genetic

information. At each end of the string are

telomeres; protective caps that keep DNA in

order as the cell replicates. Each time cells

replicate, telomeres shorten, until there is no

telomere left and the cell dies. There is a

direct link between telomere length and

aging.

Omega-3s slow cell aging

In this telomere study, 33 older adults with

mild cognitive impairment took the omega-3s

EPA and DHA, or the omega-6 linoleic acid.

Doctors gave a high dose of EPA with a low

dose of DHA, or the reverse. The daily doses

were 1,670 mg EPA plus 160 mg DHA,

1,550 mg DHA plus 400 mg EPA, or 2,200

mg linoleic acid.

After six months, doctors saw an overall

trend toward shorter telomeres, but the

omega-6 group had the most shortening.

Also, in the high-DHA group, as DHA levels

increased, telomere shortening decreased.

Doctors said these findings build on recent

reports that omega-3s appear to slow the

telomere shortening that occurs with age.

Reference: Nutrition; September, 2013,

Published Online

Vitamin K1 may improve memory

New evidence suggests vitamin K1 (phyllo-

quinone) may play a role in cognition. In this

study, doctors measured vitamin K1 in 320

men and women, aged 70 to 85, who were

not cognitively impaired at the time they

took several cognitive tests.

Researchers found no differences in making

decisions or the speed of processing informa-

tion. But those with higher levels of vitamin

K1 had better episodic memory; remember-

ing events in space and time, such as where

you left your keys. Doctors said that vitamin

K1 may play a role in the type of memory

important in everyday life.

Reference: Neurobiology of Aging; 2013, Vol.

34, No. 12, 2777-83

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